A 12-lead electrocardiogram (ECG) was obtained at the time of admission ( Figure 1).
Severe bilateral pulmonary congestion was noted on chest radiography. Initial laboratory evaluation revealed an elevated potassium level of 9.0 mmol/L and a creatinine level of 2.8 mg/dL. There is a predictable EKG progression as the serum potassium becomes more. Tall tented T waves (seen across the precordial leads) Prolonged PR segment Moderate (6.5 7.5mmol). Physical examination was remarkable for an elevated jugular venous pressure, bilateral pulmonary basilar crackles, and lower extremity pitting edema. In most cases, EKG changes have good correlation with the degree of hyperkalemia. ECG Changes in Hyperkalaemia Mild (5.5 6.5mmol). Recognition of the ECG/EKG changes of hyperkalemia can save lives. ventricular tachycardia or ventricular fibrillation and torsade de pointes.
It produces predictable changes on the ECG/EKG. Vital signs on admission were significant for hypoxia with an oxygen requirement of 4 L/min, blood pressure of 130/79 mm Hg, and heart rate of 90 bpm. Hyperkalemia (serum K+ > 5.5 mmol/l) is a life-threatening medical emergency. He denied any chest pain, palpitations, or syncope. A man in his 60s with a history of end-stage renal disease status post–renal transplant, atrial fibrillation, and sick sinus syndrome status after dual-chamber pacemaker placement presented to the emergency department complaining of progressively worsening dyspnea and diarrhea of 1 week’s duration.
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